Mneme, you immediately assigned autism with industrial society, that's a stretch.
Actually, it's not a stretch at all. This debate is nuanced, so think critically about the things I'm about to say.
This is what the NAA freaked out about, rather than the actual problem...
http://www.nationalautismassociation.org/thimerosal.php
The United States is in the midst of a tragic epidemic of autism. An analysis of the US Department of Education data from 1992-1993 in comparison to 2000-2001 indicates that there has been an average increase of 644% among all US children. In addition, 13 states have reported an almost infinite or infinite increase in autism from 1992-1993 in comparison to 2000-2001. A review of children in US schools indicates that approximately 1 in 9 children in the US is currently disabled by the US Department of Education Statistics (see attachment). Recent studies in the Journal of the American Medical Association and Pediatrics have confirmed the autism epidemic is real and not due to changes in diagnosis, populational changes nor is it explained by other factors.
At the same time that the incidence of autism was growing, the number of childhood vaccines containing thimerosal was growing, increasing the amount of ethylmercury to which infants were exposed threefold.
But here's the actual problem (The NAA was distracted by lobbies, on purpose, I'll explain momentarily, by bolding the relevant points, because they're already made)
http://www.epa.gov/hg/effects.htm
Methylmercury effects
For fetuses, infants, and children, the primary health effect of methylmercury is impaired neurological development. Methylmercury exposure in the womb, which can result from a mother's consumption of fish and shellfish that contain methylmercury, can adversely affect a baby's growing brain and nervous system. Impacts on cognitive thinking, memory, attention, language, and fine motor and visual spatial skills have been seen in children exposed to methylmercury in the womb. Recent human biological monitoring by the Centers for Disease Control and Prevention in 1999 and 2000 (PDF) (3 pp., 42 KB, About PDF) shows that most people have blood mercury levels below a level associated with possible health effects. More recent data from the CDC support this general finding.
Outbreaks of methylmercury poisonings have made it clear that adults, children, and developing fetuses are at risk from ingestion exposure to methylmercury. During these poisoning outbreaks some mothers with no symptoms of nervous system damage gave birth to infants with severe disabilities, it became clear that the developing nervous system of the fetus may be more vulnerable to methylmercury than is the adult nervous system.
For more information on fish consumption advisories across the country, visit EPA's fish consumption web pages.
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http://www.ncbi.nlm.nih.gov/pubmed/20813099
Effects of low level of methylmercury on proliferation of cortical progenitor cells.
Xu M, Yan C, Tian Y, Yuan X, Shen X.
Source
XinHua Hospital affiliated to Shanghai Jiao Tong University School of Medicine, Shanghai Institute for Pediatric Research, Shanghai Key Laboratory of Children's Environmental Health, Shanghai 200092, China.
Abstract
Methylmercury (MeHg) is a potent environmental neurotoxin that shows toxicity to developing central nervous system (CNS), causing brain damage in children even at low exposure levels. However, the mechanisms for its effect on CNS are not well understood. In current study, primary cultures of progenitor cells from embryonic cerebral cortex were used as a model system to study the potential effect and the underlying mechanism of MeHg on neural progenitor cells. Results showed that, in cultured cortical progenitor cells, 48-h exposure to low-level of MeHg (at 2.5 nM, 5 nM and 50 nM, respectively) caused G1/S cell cycle arrest in a dose-dependent manner without inducing cell death. Interestingly, the expression of cyclin E, which promotes G1/S transition, but not cyclin D1 and CDK2, was selectively downregulated by exposure of MeHg. In addition, low-level of MeHg inhibited the maintenance of ERK1/2 phosphorylation, possibly by abolishing the late phase ERK1/2 activation induced by bFGF. Thus, MeHg may induce proliferation inhibition and cell cycle arrest of neural progenitor cells via regulating cyclin E expression and perturbing a pathway that involves ERK1/2.
Copyright © 2010 Elsevier B.V. All rights reserved.
PMID:
20813099
[PubMed - indexed for MEDLINE]
and how we know all of this
http://youtu.be/ihFkyPv1jtU
http://www.ec.gc.ca/mercure-mercury/default.asp?lang=En&n=F111AAC6-1
Disposing of Mercury Products
Mercury is found in some consumer and industrial products and can be released into the environment when a product is disposed of improperly. Mercury then enters the global mercury cycle and can bioaccumulate in living organisms, potentially affecting ecosystems and human health. Find out which products contain mercury and ensure that they are recycled or disposed of properly.
The fate of the mercury contained in various products depends upon the method of disposal at the end of the product's life. The more fragile products, such as fluorescent lamps, may break during transportation and release mercury into the air. The US Environmental Protection Agency estimates that 3% of the total mercury in discarded fluorescent lamps is released to the atmosphere during transportation to a disposal facility, while other researchers estimate emissions are as high as 17%. If a mercury-containing product ends up in a landfill, the mercury can leach into the surrounding soil or be released into the atmosphere. If waste is incinerated, the amount of mercury released into the atmosphere may be higher. Without any pollution controls, almost all of the mercury entering an incinerator will be emitted with the flue gas.
Ensuring that mercury-containing products are properly and safely recycled or disposed of is one step that can be taken to protect environmental and human health. To avoid the risk associated with disposal of mercury-containing products, purchase mercury-free alternatives if available. Please visit the What Can I Do? page for more suggestions on how to reduce mercury releases to the environment.
...and where it comes from...
http://www.ec.gc.ca/mercure-mercury/default.asp?lang=En&n=72C0CC0E-1
Products that Contain Mercury
Mercury has been used in a variety of consumer and commercial products because it exhibits properties of both a metal and a liquid at room temperature, it is a good conductor of electricity, and it reacts precisely to temperature and pressure changes. Unfortunately, when these products are broken and the spilled mercury poured down the drain or when these products are disposed of in landfill sites, the mercury can become a part of the global mercury cycle. Mercury releases from products, as they break down in both active and closed landfills, may represent a significant pathway for the transport and eventual deposition of the toxin in various terrestrial and aquatic ecosystems. For example, the amount of mercury contained in a typical fluorescent tube (approximately 20 milligrams) is sufficient to contaminate around 20 000 litres of water beyond Health Canada limits for safe drinking water (0.001 milligrams of mercury per litre of water). In general, mercury may be released from municipal solid waste landfills as a trace component of landfill gas, which is generated during the decomposition of waste under anaerobic conditions, or in the liquid leachate flowing from the site.
...and where it ends up...
http://water.usgs.gov/wid/FS_216-95/FS_216-95.html
How does mercury become a toxicological problem?
Like many environmental contami-nants, mercury undergoes bioaccumulation. Bioaccumulation is the process by which organisms (including humans) can take up contaminants more rapidly than their bodies can eliminate them, thus the amount of mercury in their body accumulates over time. If for a period of time an organism does not ingest mercury, its body burden of mercury will decline. If, however, an organism continually ingests mercury, its body burden can reach toxic levels. The rate of increase or decline in body burden is specific to each organism. For humans, about half the body burden of mercury can be eliminated in 70 days if no mercury is ingested during that time. Biomagnification is the incremental increase in concentration of a contaminant at each level of a food chain
Figure 4. Mercury (Hg) biomagnifies from the bottom to the top of the food chain. Even at very low input rates to aquatic ecosystems that are remote from point sources, biomagnification effects can result in mercury levels of toxicological concern.
This phenomenon occurs because the food source for organisms higher on the food chain is progressively more concentrated in mercury and other contaminants, thus magnifying bioaccumulation rates at the top of the food chain. The bioaccumulation effect is generally compounded the longer an organism lives, so that larger predatory game fish will likely have the highest mercury levels. Adding to this problem is the fact that mercury concentrates in the muscle tissue of fish. So, unlike organic contaminants (for example PCBs and dioxins) which concentrate in the skin and fat, mercury cannot be filleted or cooked out of consumable game fish.
What are the human health effects of mercury toxicity?
Humans generally uptake mercury in two ways: (1) as methylmercury (CH3Hg+) from fish consumption, or (2) by breathing vaporous mercury (Hg0) emitted from various sources such as metallic mercury, dental amalgams, and ambient air. Our bodies are much more adapted for reducing the potential toxicity effects from vaporous mercury, so health effects from this source are relatively rare. Methylmercury, on the other hand, affects the central nervous system, and in severe cases irreversibly damages areas of the brain
...and how bioaccumulation works...
http://toxics.usgs.gov/definitions/bioaccumulation.html
Bioaccumulation
Definitions
Bioaccumulation – “The biological sequestering of a substance at a higher concentration than that at which it occurs in the surrounding environment or medium.” - U.S. Geological Survey, 2007
Bioaccumulation – “Bioaccumulation (or bioconcentration) is the uptake of organic compounds by biota from either water or food. Many toxic organic chemicals attain concentrations in biota several orders of magnitude greater than their aqueous concentrations, and therefore, bioaccumulation poses a serious threat to both the biota of surface waters and the humans that feed on these surface-water species.” – Smith and others, 1988
Bioaccumulation – “General term describing a process by which chemicals are taken up by an organism either directly from exposure to a contaminated medium or by consumption of food containing the chemical.” – U.S. Environmental Protection Agency, 2010
Bioaccumulation – “Bioaccumulation is defined as the accumulation of chemicals in the tissue of organisms through any route, including respiration, ingestion, or direct contact with contaminated water, sediment, and pore water in the sediment.” – U.S. Environmental Protection Agency, 2000
Bioaccumulation – “Progressive increase in the amount of a substance in an organism or part of an organism which occurs because the rate of intake exceeds the organism’s ability to remove the substance from the body.” – International Union of Pure And Applied Chemistry, 1993
Related Definitions
Bioaccumulation Factor (BAF) – “The ratio of the contaminant in an organism to the concentration in the ambient environment at a steady state, where the organism can take in the contaminant through ingestion with its food as well as through direct content.” – U.S. Environmental Protection Agency, 2010
Bioaccumulation Factor (BAF) – “The bioaccumulation factor (also called the BAF) is analogous to the BCF [bioconcentration factor], but applies to field measurements or to laboratory measurements with multiple exposure routes. The BAF is the ratio of contaminant concentration measured in biota in the field (or under multiple exposure conditions) to the concentration measured in the surrounding water.” – Nowell and others, 1999
Bioconcentration Factor (BCF) – “A major concern for environmental contamination is the extent to which pollutants concentrate from water into aquatic organisms such as fish. The extent of such concentration, termed the bioconcentration factor (BCF), is given by the ratio of the pollutant concentration in fish to that in water.” – Chiou, 2002
...and how bad the problem is already...
http://www.usgs.gov/themes/factsheet/146-00/fig4b.gif
http://www.epa.gov/hg/about.htm
Forms of mercury. Mercury is a naturally occurring element that is found in air, water and soil. It exists in several forms: elemental or metallic mercury, inorganic mercury compounds, and organic mercury compounds. More information
Sources of mercury. Mercury is an element in the earth's crust. Humans cannot create or destroy mercury. Pure mercury is a liquid metal, sometimes referred to as quicksilver that volatizes readily. It has traditionally been used to make products like thermometers, switches, and some light bulbs.
Mercury is found in many rocks including coal. When coal is burned, mercury is released into the environment. Coal-burning power plants are the largest human-caused source of mercury emissions to the air in the United States, accounting for over 50 percent of all domestic human-caused mercury emissions (Source: 2005 National Emissions Inventory). EPA has estimated that about one quarter of U.S. emissions from coal-burning power plants are deposited within the contiguous U.S. and the remainder enters the global cycle. Burning hazardous wastes, producing chlorine, breaking mercury products, and spilling mercury, as well as the improper treatment and disposal of products or wastes containing mercury, can also release it into the environment. Current estimates are that less than half of all mercury deposition within the U.S. comes from U.S. sources. More information.
Sources of mercury compounds. In the U.S., mercury compounds are manufactured in small amounts for specialty uses, such as chemical and pharmaceutical applications. Larger quantities of these compounds are generated as byproducts from pollution control activities at gold mines or in waste. Elemental mercury is processed in the U.S. from byproduct mercury compounds, and an unknown quantity of mercury compounds is imported into the United States for conversion to elemental mercury. Learn more about mercury compounds (PDF). (123 pp, 738K, About PDF)
Exposure to mercury. Mercury in the air eventually settles into water or onto land where it can be washed into water. Once deposited, certain microorganisms can change it into methylmercury, a highly toxic form that builds up in fish, shellfish and animals that eat fish. Fish and shellfish are the main sources of methylmercury exposure to humans. Methylmercury builds up more in some types of fish and shellfish than others. The levels of methylmercury in fish and shellfish depend on what they eat, how long they live and how high they are in the food chain.
EPA works with the U.S. Food and Drug Administration (FDA) and with states and tribes to issue advice to women who may become pregnant, pregnant women, nursing mothers and parents of young children about how often they should eat certain types of commercially-caught fish and shellfish. Fish advisories are also issued for men, women, and children of all ages when appropriate. In addition, EPA releases an annual summary of information on locally-issued fish advisories and safe-eating guidelines to the public. Fish is a beneficial part of the diet, so EPA & FDA encourage people to continue to eat fish that are low in methylmercury. More information
Another less common exposure to mercury that can be a concern is breathing mercury vapor. These exposures can occur when elemental mercury or products that contain elemental mercury break and release mercury to the air, particularly in warm or poorly-ventilated indoor spaces. More information
Health effects of mercury. Mercury exposure at high levels can harm the brain, heart, kidneys, lungs, and immune system of people of all ages. Research shows that most people's fish consumption does not cause a health concern. However, it has been demonstrated that high levels of methylmercury in the bloodstream of unborn babies and young children may harm the developing nervous system, making the child less able to think and learn. More information
Ecological effects of mercury. Birds and mammals that eat fish are more exposed to mercury than other animals in water ecosystems. Similarly, predators that eat fish-eating animals may be highly exposed. At high levels of exposure, methylmercury's harmful effects on these animals include death, reduced reproduction, slower growth and development, and abnormal behavior. More information
Reducing mercury releases. EPA issues regulations that require industry to reduce mercury releases to air and water and to properly treat and dispose of mercury wastes. In 2010, EPA is working to develop emissions standards for power plants under Clean Air Act section 112, consistent with the D.C. Circuit’s February 2008 opinion (PDF) (18pp, 51k, about PDF) regarding the Clean Air Mercury Rule (CAMR). On October 6, 2009, EPA published a final rule that limits emissions, including emissions of mercury, from medical waste incinerators.
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http://www.bitsofscience.org/mercury-extinction-permian-triassic-polution-4617/
Mercury: a new culprit in end-Permian mass extinction event
Posted on January 6, 2012 by Jorn van Dooren
Historical variations of mercuryVolcanic eruptions have already been appointed as the main culprit of the Permian-Triassic mass extinction.
Previous research indicated that the resulting rise in atmospheric and oceanic carbon lead to the Great dying. But new findings in the journal Geology point to a large influx of mercury as a another cause likely involved in the annihilation.
The largest source of mercury on Earth are volcanic eruptions. But while the end-Permian was a period with the highest volcanic activity in Earth’s history, no one before had thought of looking into mercury as a potential contributing factor to the greatest mass-extinction in the history of our planet.
Too much to clean up
Normally algae act as the vacuum cleaners of the ocean, absorbing mercury and transporting it to the ocean floor as they die. But the estimated amount of mercury inserted into the ecosystem by the end-Permian volcanic eruptions was thirty times higher than current levels. A load so overwhelming that the natural buffering system of algae could not stop it from spreading through the entire ecosystem. With a substance as toxic as mercury such levels would have been catastrophic for the already stressed global ecosystem.
The accumulation of stress factors on the marine ecosystems eventually led to the extinction of approximately 90 per cent of all aquatic life. But the cascading events during the end-Permian mass extinction also led to anoxic marine conditions. Which may in fact have been a blessing in disguise, since the absence of oxygen in the oceans in fact made the chemical drawdown of mercury sulphides possible, that sank to the ocean floor, thereby removing mercury from the ecosystem.
The blessing of euxinic conditions
The remaining 10 per cent of ocean dwelling species encompassed all of Life’s Kingdoms which eventually, after millions of years, managed to recover. Without the anoxic period however, this might not have been possible due to remaining high levels of mercury.
While mercury levels were exceptionally high during the mass extinction event, similar levels can currently be found at certain areas on Earth. The difference however is that those areas are contaminated through human activity. The most notable of such areas are ponds near smelters, which are so highly contaminated that aquatic life is nearly impossible. But also coal-fired power plants are a source of increased mercury levels in their direct surroundings.
The research team warns about the continuing introduction of mercury into the environment through industrial emissions. They plan to take a closer look at mercury levels during other mass extinction events, to see if there is a significant rise in those periods as well and possibly to discover what the effect of rising mercury levels on the Holocene mass extinction might be.
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http://www.tulane.edu/~bfleury/envirobio/enviroweb/DeadZone.htm
The Dead Zone
The Gulf of Mexico hypoxic zone is a seasonal phenomena occurring in the northern Gulf of Mexico, from the mouth of the Mississippi River to beyond the Texas border. It is more commonly referred to as the Gulf of Mexico Dead Zone, because oxygen levels within the zone are too low to support marine life. The Dead Zone was first recorded in the early 1970's. It originally occurred every two to three years, but now occurs annually. In the summer of 1999 the Dead Zone reached its peak, encompassing 7,728 square miles.
Hypoxic conditions arise when dissolved oxygen levels in the water fall below two milligrams per liter of water, too low to sustain animal life in the bottom strata of the ocean. The Dead Zone forms each spring as the Mississippi and Atchafalaya Rivers empty into the Gulf, bringing nutrient rich waters that form a layer of fresh water above the existing salt water. It lasts until late August or September when it is broken up by hurricanes or tropical storms. The nutrients provide favorable conditions for excessive growth of algae that utilize the water’s oxygen supply for respiration and when decomposing.
The Mississippi River Basin covers forty-one percent of the continental United States, contains forty-seven percent of the nation’s rural population, and fifty-two percent of U.S. farms. The waste from this entire area drains into the Gulf of Mexico through the Mississippi River. Included in this agricultural waste are phosphorus and nitrogen, the primary nutrient responsible for algal blooms in the Dead Zone. Nitrogen and phosphorus were first used in fertilizers in the United States in the 1930s. Concentrations of nitrate and phosphate in the lower Mississippi have increased proportionately to levels of use of fertilizers by agriculture since the 1960s, when fertilizer use increased by over two million metric tons per year. Overall, nitrogen input to the Gulf from the Mississippi River Basin has increased between two and seven times over the past century. In addition to agricultural waste, inadequately treated or untreated sewage and other urban pollution is also dumped into these waters. Nitrogen is normally a limiting factor, meaning its restricted quantities limit plant growth and reproduction. However, excessive amounts of nitrogen lead to eutrophication, the takeover of nutrient-rich surface water by phytoplankton or other plants. If nutrient pollution is not greatly reduced, fish and shellfish may someday be permanently replaced by anaerobic bacteria.
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http://www.ncbi.nlm.nih.gov/pubmed/3553530
Mercury methylation by bacteria.
Trevors JT.
Abstract
Bacteria capable of methylating Hg2+ have been isolated from sediment, water, soil and the gastrointestinal tract of humans. However, very little is known about the physiology and genetics of the mechanisms controlling Hg2+ methylation. Mercury methylation can be either chromosomal or plasmid-encoded in bacteria. In addition, the extent of nonbiological methylation is not well understood in environmental samples, where there are numerous physical, chemical and biological factors that control the methylation process. It is known that methylation of Hg2+ is mediated by a series of enzymatic reactions that are also responsible for the anaerobic evolution of methane. However, under highly reduced environments the reaction can also occur nonbiologically. It is possible that certain bacteria use methylation as a resistance/detoxification mechanism.
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http://www.nicholas.duke.edu/wetland/vaith_96_est.htm
Abstract. Elevated mercury concentrations have been reported in fish and wildlife from the Everglades in recent years. The hypothesis that eutrophication caused by the impact of phosphorus- (P) rich agricultural runoff stimulated methylmercury accumulation was put forward because eutrophication had been shown to be a cause for methylmercury accumulation in other ecosystems. We tested this hypothesis by obtaining total mercury (HgT) concentrations and accumulation rates and estimating the potential for microbial methylation and methylmercury degradation in peat soils collected along a P gradient in Water Conservation Area 2A (WCA-2A). A negative correlation observed between HgT and P concentrations in soils (r2 = 0.64) was explained by increased peat accretion rates in a nutrient-enriched area (7.1-7.55 mm yr–1) as compared to an unenriched area (1.92-2.50 mm yr–1), estimated using ead-210 and cesium-137 dating. Total Hg accumulation rates (post-1964) were comparable for the enriched and unenriched sites (29-30 and 29-37 m g m–2 yr–1, respectively). Thus, calculations of HgT accumulation rates are confounded by differences in peat accretion rates in the Everglades. Potential rates for both methylation (2.3-48.6 ng g–1 day–1) were and demethylation (6.5-113.2 ng g–1 day–1) were higher in samples from WCA-2A than in samples collected in an area of the Everglades that had never been exposed to nutrients. However, trends suggesting the relationships of these activities to the P gradient in WCA-2A were not detected, and methylation to demethylation ratios did not correlate with soil P concentrations. The results suggest that (i) nutrient-enriched agricultural runoff originating upstream of WCA-2A did not contribute significantly to HgT built up in the northern Everglades and (ii) eutrophication did not affect the potential the potential for net methylmercury formation in peat soils.
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http://www.wri.org/project/eutrophication/map
...so.... to reiterate....
Mneme, you immediately assigned autism with industrial society, that's a stretch.
but the EPA says...
Mercury is found in many rocks including coal. When coal is burned, mercury is released into the environment. Coal-burning power plants are the largest human-caused source of mercury emissions to the air in the United States, accounting for over 50 percent of all domestic human-caused mercury emissions (Source: 2005 National Emissions Inventory).
So it's not a stretch, it's a matter of knowing what I'm talking about. This is the seventh post like this I've made to these forums, if you have evidence contrary to what I'm saying, from authoritative reliable sources, backed by extensive research (as I have repeatedly provided for my statements here)... I'll hear the counterclaim.
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Murdoch is an elitist, but one more hooked into public opinion, because his income (what he cares most about) is connected with public perception.
well...
http://youtu.be/V4LFCfv2b6U
http://en.wikipedia.org/wiki/Operation_Mockingbird
In 1948, Frank Wisner was appointed director of the Office of Special Projects (OSP). Soon afterwards OSP was renamed the Office of Policy Coordination (OPC). This became the covert action branch of the Central Intelligence Agency. Wisner was told to create an organization that concentrated on "propaganda, economic warfare; preventive direct action, including sabotage, anti-sabotage, demolition and evacuation measures; subversion against hostile states, including assistance to underground resistance groups, and support of indigenous anti-Communist elements in threatened countries of the free world."[3]
Later that year Wisner established Mockingbird, a program to influence foreign media. Wisner recruited Philip Graham from The Washington Post to run the project within the industry. According to Deborah Davis in Katharine the Great; "By the early 1950s, Wisner 'owned' respected members of The New York Times, Newsweek, CBS and other communications vehicles."[4] Wisner referred to this apparatus as a "Mighty Wurlitzer", referencing the theater organ capable of controlling diverse pipes, instruments, and sound effects from a central console.[5]
In 1951, Allen W. Dulles persuaded Cord Meyer to join the CIA. However, there is evidence that he was recruited several years earlier and had been spying on the liberal internationalist organizations he had been a member of in the later 1940s.[6] According to Deborah Davis, Meyer became Mockingbird's "principal operative".[7]
In 1977, a People article by Alexander Butler alleged that one of the most important journalists under the control of Operation Mockingbird was Joseph Alsop, whose foreign affairs articles appeared in over 300 different newspapers. Other journalists alleged by People Magazine to have been willing to promote the views of the CIA included Stewart Alsop who headed the international bureau of New York Herald Tribune, Ben Bradlee foreign affairs correspondent for Newsweek, James Reston for the international section of the New York Times, Charles Douglas Jackson foreign photo-journalist for Time Magazine, and international correspondents such as Walter Pincus of the Washington Post, Charles Bartlett of the Chattanooga Times and William C. Baggs and Herb Gold of The Miami News.[9] According to Nina Burleigh (A Very Private Woman), these journalists sometimes wrote articles that were commissioned by Frank Wisner. The CIA also provided them with classified information to help them with their work.[10]
Congressional hearings in 1976 proved CIA had been paying of editors and reporters in most mainstream media outlets
http://en.wikipedia.org/wiki/News_Corporation
News Corp was created in 1979 by Rupert Murdoch as a holding company for News Limited. News Limited was created by Murdoch from the assets he inherited in 1952 following the death of his father, Sir Keith Murdoch, and subsequent expansion. The main asset left to him was ownership of the Adelaide afternoon tabloid, The News. News Limited operates today as News Corporation's Australian brand, operating out of Surry Hills, in Sydney.
News Ltd. made its first acquisition in the United States in 1973, when it purchased the San Antonio Express-News. Soon afterwards it founded the National Star, a supermarket tabloid, and in 1976 it purchased the New York Post.
It's a "sticky wicket" of an issue. Let's skip the rest of the intentionally obscured history of propaganda empires.
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But under this corporatist crap, we are running into an issue where money is getting killed.
Good! Bring on the post-scarcity environment! To hell with classism, to hell with empires, to hell with poverty and starvation and lobbyism and totalitarian economies. Screw them all!
We can, have, and will... do better than this.
